“It’s inappropriate to immunize in the arm. We have known that for 40 odd years.”
“The vaccines cannot work in principle.”
People who had tonsillectomies or adenoidectomies as a child had a 2-3 times greater risk of upper respiratory tract diseases.
I watched the clip above recently on Couey’s channel.
It’s a short segment where Prof. Edward “Ted” Steele, an Australian, is talking about what has been known about mucosal immunity and vaccination “…for 40 odd years.”
In doing so he refers to another prominent Australian, Prof. Robert Clancy, a mucosal immunologist. There’s a video of him talking about the subject below.
I will summarise the point using my layman’s language:
Respiratory infections require immunity at source. In the airway.
We require an immune response in the mucosa of the airway; it’s called mucosal immunity.
Generating antibodies in the blood, as covid vaccines do, “…cannot work in principle…” to fight a virus in the airway.
This principle applies to flu and whooping cough vaccines.
Let’s catch our breath here for a moment.
Are we saying that they vaccinated 5 billion people, multiple times, with an injection that is tasked with generating antibodies in the blood, that “cannot work in principle” to combat covid, a respiratory virus, and that they always knew it wouldn’t work?
Yes.
Girardot wrote about this back in Jan 2022, referencing Steele:
How can an injection in the deltoid stimulate an immunity in the mucus?
Respiratory virus like SARS-COV-2 typically propagate in the mucus: mouth, nose, digestive tract and lungs. For propagation to be stopped in the mucus, notably in the lungs, a preemptive immune arsenal needs to be stimulated there. This is exactly what occurs once recovered from a natural infection:
a sterilising immunity is provided by potent resident memory T and B-cells - along with neutralising IgA antibodies - that are positioned in large numbers as a sentinel force to kill in-the-egg any starting infection.
I have addressed this at length in my June article comparing natural immunity and vaccine-induced immunity as well as in my August article on pre-existing immunity. I am not alone in thinking along these lines; many renowned scientists share a similar perspective that intramuscular vaccines cannot work for mucosal viruses:
Professor Sucharit Bhakdi in Germany in a recent article titled “ Why intramuscular COVID-19 vaccination must fail ” made that argument (see video).
Professor Edward J. Steele of Australia in a recent interview on Asia Pacific Today titled “The Origins Of Covid-19 & Why The Vaccines Don't Work” also made the same point.
Professor Michael W. Russell in the US hinted the same thing in an article “Mucosal Immunity in COVID-19: A Neglected but Critical Aspect of SARS-CoV-2 Infection”.
Already in 1992, McGhee et al in an article titled “The mucosal immune system: from fundamental concepts to vaccine development” had pointed to this fallacy.
“It is surprising that despite our current level of understanding of the common mucosal immune system, almost all current vaccines are given to humans by the parenteral route. Systemic immunisation is essentially ineffective for induction of mucosal immune responses.” McGhee et al, 1992
Girardot puts it this way, as only he can:
Imagine you want to catch someone when he gets out of a building, and he never does: he’s actually using tunnels from one building to the next. Well these vaccines are essentially useless because not only is the virus using tunnels, but you’re not even in the same town!
The virus is inside a Mucustown building while antibodies are outside a building in Bloodville!
Looks like a missed meeting to me. What do you think? …
Girardot, to my knowledge, is the only person that wrote about the impact of masks disrupting the smooth circular vortex flow of air within the airway, that maximizes the chance for pathogens to pass over the mucosa. We have been built with the most wonderful technology to deal with airborne pathogens, and everything we have done has interfered and disabled our natural ability to respond.
We Are All Already Wearing A Mask... - by Marc Girardot (substack.com)
I wrote recently about the ineffectiveness of flu vaccines.
How to keep vaccine factories open. - Lies are Unbekoming (substack.com)
I’m now wondering how much of that ineffectiveness is due to this blood vs mucosa, location issue. It’s likely to be a primary cause. When Steele says that they have known for 40 odd years, I suspect he is referring directly to the flu vaccine sham.
Steele also mentions whooping cough (pertussis), the “P” in DTP, with the same dynamic going on.
Steele makes another very important point, which is that anti-bodies generated in the mucosa are non-inflammatory while anti-bodies generated in the blood are pro-inflammatory.
How many different ways can these people lie to us? How many different ways are there to take advantage of the trusting and the gullible. Plenty, it seems.
Steele mentions gut associated lymphoid tissue activation, or GALT1. See the footnotes for some sciency stuff on that and airway-associated lymphoid tissue activation (AALT).
Steele starts with an anti-pharma posture but them seamlessly transitions into a pro-pharma posture, at which point Couey rightly pauses the video and chips him for it.
Now, just before I move on, I want to dwell on this point a bit longer.
We all know by now that the covid vaccines don’t work. Many of us have known that from the early days. Most of the talk has been about their dangers, and rightly so. But the point being made here, is so staggering, that I am choosing to dwell on it a bit. Not only do they not work, not only did they not work from the start, but…
THEY WERE NEVER GOING TO WORK…..and…..THEY KNEW IT.
Now, let me say it again, a bit calmer and without the caps.
They were never going to work, and they knew it.
They are all risk, and there was never going to be any benefit.
Ok, let’s move on…to tonsils.
Tonsils are a major part of the first stage “hardware” located in the airway and built to generate mucosal immunity against respiratory pathogens.
Recently someone I know, in passing, said that they were scheduled to take their 3-year-old boy in for surgery to remove his tonsils, as his tonsils were causing him sleeping problems.
My ears pricked as the subject of tonsils and their removal has been on my mind recently. I have now come to see as highly suspect any surgery where a body part is removed. My default posture now to all “removal” is that there is likely a collection of untruths, lies and incentives that have led up to the surgery.
So, I sent this young father the following email:
Hi John
Here is some information for you guys to consider in your decision, and/or discuss with your doctor.
This I believe is the biggest and most recent meta-analysis of the long-term impacts of removing part of the immune system hardware.
In this population-based cohort study of almost 1.2 million children, removal of adenoids or tonsils in childhood was associated with significantly increased relative risk of later respiratory, allergic, and infectious diseases. Increases in long-term absolute disease risks were considerably larger than changes in risk for the disorders these surgeries aim to treat.
--
A total of up to 1 189 061 children were included in this study (48% female); 17 460 underwent adenoidectomy, 11 830 tonsillectomy, and 31 377 adenotonsillectomy; 1 157 684 were in the control group. Adenoidectomy and tonsillectomy were associated with a 2- to 3-fold increase in diseases of the upper respiratory tract.
This looks specifically at the issue of sleep.
Authors' conclusions:
In otherwise healthy children, without a syndrome, of older age (five to nine years), and diagnosed with mild to moderate OSAS by PSG, there is moderate quality evidence that adenotonsillectomy provides benefit in terms of quality of life, symptoms and behaviour as rated by caregivers and high quality evidence that this procedure is beneficial in terms of PSG parameters. At the same time, high quality evidence indicates no benefit in terms of objective measures of attention and neurocognitive performance compared with watchful waiting. Furthermore, PSG recordings of almost half of the children managed non-surgically had normalised by seven months, indicating that physicians and parents should carefully weigh the benefits and risks of adenotonsillectomy against watchful waiting in these children. This is a condition that may recover spontaneously over time.
These are two good reads also.
New study reveals long-term risks of tonsillectomy - Lown Institute
Hope this helps.
Regards
Unbekoming
He, and his wife, emailed me afterwards, very thankful for the information, but they went ahead with the removal of the 3-year old’s tonsils.
Now, I am not a tonsils expert, frankly I’m not an expert on any of the subjects I write about, but if someone’s tonsils are regularly swollen, wouldn’t the right thing to do be to go after the “cause” of the swelling, rather than just chop out the hardware?
If my arm is regularly infected and swollen, and preventing me from sleeping properly, would it make sense that on my third visit in a 12-month period, to my doctor, I am advised to chop off my arm? At the age of three…!!!
I wonder if there is any connection between childhood vaccination and tonsillitis. I’m happy to wager that there is. If anyone knows anything about that, please let me know.
I wrote about “intelligence” and “design” yesterday, and I think it’s fair to say that this “chop off and remove” paradigm is a child of the mechanistic worldview.
This causes that. If we remove this, we fix that.
An infantile posture to the wonder, beauty and complexity that is the human body. If you had a “design” paradigm, I think you would find it much harder to just “chop” things out. The hubris to do that would likely scurry away, into the shadows, with its tail between its legs.
Also, this from 2003 about tonsillectomy:
In a Cochrane systematic review, we found no good evidence for or against tonsillectomy. Evidence from two randomised controlled trials (RCTs) in children was inconclusive.
--
Despite this absence of “evidence”, tonsillectomy remains popular with both ENT surgeons and, more particularly, their patients.
Anyway, the JAMA long-term study (with 1.2m kids) basically settles the issue for me. It is not without material long-term consequences.
Adenoidectomy and tonsillectomy were associated with a 2- to 3-fold increase in diseases of the upper respiratory tract.
You don’t get to just remove a body part, especially one as important and the tonsils and simply shrug and say, “don’t worry, you can do without them…they don’t do much anyway”.
Here is a “fact sheet” from a major Sydney children’s hospital:
Tonsillectomy | Sydney Children's Hospitals Network (nsw.gov.au)
What is most interesting is what is NOT said. There is nothing in the “fact sheet” that explains the long-term consequences of tonsil removal. In their framing there is only benefit, and no cost.
Simply unbelievable.
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GALT and AALT
Gut-associated lymphoid tissue (GALT) activation refers to the immune response that occurs in the lymphoid tissue lining the gut. This tissue is rich in immune cells, such as T cells and B cells, and is responsible for protecting the body from pathogens that enter through the gut. Activation of GALT can occur in response to an infection or other inflammatory stimulus, and results in the proliferation and activation of immune cells, as well as increased production of antibodies. The end result is an immune response that helps to clear the pathogen and restore the gut to its normal state.
The airways also have immune cells and lymphoid tissue that help protect the body from pathogens. This is known as the airway-associated lymphoid tissue (AALT). Activation of AALT, also called airway immune response, occurs in response to an infection or other inflammatory stimulus in the airways, such as allergens or pollutants. This results in the proliferation and activation of immune cells, as well as increased production of antibodies and other immune molecules. This immune response helps to clear the pathogen and restore the airways to their normal state. In addition to infections, conditions such as asthma and chronic obstructive pulmonary disease (COPD) are characterized by chronic activation of AALT.
Respiratory viruses are one of the common triggers for airway-associated lymphoid tissue (AALT) activation. When a respiratory virus such as the common cold or influenza enters the body through the nose or mouth, it can infect the cells lining the airways. This initiates an immune response in the AALT, which includes the activation and proliferation of immune cells such as T cells and B cells. These immune cells help to clear the virus and restore the airways to their normal state.
Tonsils are part of the airway-associated lymphoid tissue (AALT) and play a role in the immune response to respiratory viruses. The tonsils are clusters of lymphoid tissue located at the back of the throat and are part of the body's immune system. They are particularly rich in T cells and B cells, which are important in fighting off infection.
When a respiratory virus enters the body, it can infect the cells lining the airways, including the tonsils. This initiates an immune response in the tonsils, which includes the activation and proliferation of immune cells such as T cells and B cells. These immune cells help to clear the virus and restore the airways to their normal state. The immune response also results in increased production of antibodies, which provide immunity to the specific virus, and other immune molecules that help to fight off the infection. However, in some cases, the tonsils can become swollen and inflamed in response to a viral infection, a condition known as tonsillitis.
In mucosal immunity, antibodies of the IgA class are typically produced in large quantities. IgA antibodies are a type of immunoglobulin that are produced by B cells in the mucosal surfaces, such as the gut, lungs, and nasal passages. They are the most abundant antibody class in the mucosal secretions.
IgA antibodies play a critical role in protecting the body from pathogens that enter through the mucosal surfaces. They help to neutralize pathogens by binding to their antigens, thereby preventing the pathogens from infecting host cells. They also recruit other immune cells to the site of infection, such as activating complement and recruiting phagocytes, which can help to clear the pathogen from the body.
IgG and IgA are both types of immunoglobulins, also known as antibodies, that are produced by B cells and play a critical role in protecting the body from pathogens. However, there are some key differences between the two types of antibodies:
1. Location: IgG is found mainly in the blood and extracellular fluids, while IgA is found mainly in mucosal secretions such as saliva, tears, breast milk and respiratory, gastrointestinal and urogenital tracts.
2. Function: IgG is mainly responsible for providing immunity to pathogens that have entered the bloodstream, such as bacteria and viruses. IgA is mainly responsible for providing immunity to pathogens that enter through mucosal surfaces, such as the gut, airways, and reproductive tract.
3. Mechanism of action: IgG neutralizes pathogens by binding to their antigens, thereby preventing the pathogens from infecting host cells. IgA neutralizes pathogens by binding to their antigens, and also by creating a physical barrier that prevents pathogens from adhering to the mucosal surfaces.
4. Quantity: IgG is the most prevalent and long-lasting antibody in the body. IgA is the most abundant antibody class in the mucosal secretions.
5. Duration of protection: IgG antibodies provide long-term protection against pathogens, because they can remain in the body for months or even years after an infection. IgA antibodies provide shorter-term protection against pathogens, because they are cleared from the body more quickly and need to be continually produced to provide ongoing protection.
Mucosal Immunity